Editing Q-94-109A (section)

=== Membrane and intracellular processes ===
Researchers performed detailed analyses using techniques such as [[freeze fracture electron microscopy]], [[Scanning Tunneling Microscope|STM]], [[histopathology]], and various biochemical assays.<ref name="Q-94-109A"/> They discovered that [[neuroprotofibrils]] terminated at dendritic branch points, an anomaly correlated with nearby fibroblast analog activity and pathological changes at [[myoneural junction]]s.<ref name="Q-94-109A"/>

Membrane studies revealed disruptions in the normal phosphorylation processes, ion channel distributions, and mitochondrial-golgi analog (MG) concentrations near synaptic areas.<ref name="Q-94-109A"/> The hydrogen-mediated phosphorylation, crucial for energy budgeting, was most efficient where subneural clefts were shortest.<ref name="Q-94-109A"/> These technical findings contributed to understanding the pathological processes leading to [[neuropathology]] in the specimen.<ref name="Q-94-109A"/>